- intermediate transsphincteric fistulas
- high transsphincteric fistulas
- suprasphincteric fistulas
- acute abscesses with an existing fistula that cannot be primarily split
Etiologyof cryptoglandular anal fistulas
The cryptoglandular hypothesis by Parks is generally accepted as an explanation for the development of perianal abscesses and fistulas. According to this, a perianal abscess initially results from the obstruction of a proctodeal gland or its duct, followed by infection. The fistula represents the abscess drainage channel, which forms as a result of chronic inflammation and epithelialization. The internal opening of all cryptoglandular anal fistulas is located—in accordance with the proctodeal gland opening—in an anal crypt at the dentate line, whereas the external fistula opening is in the perianal skin.
The classification of cryptoglandular anal fistulas is based on their course in relation to the anal sphincter apparatus. They are distinguished as
· Subanodermal/subcutaneous anal fistula
· Intersphincteric anal fistula
· Transsphincteric anal fistula
Note: In the case of a transsphincteric anal fistula, the fistula tract penetrates the external anal sphincter and enters the ischioanal fossa. Depending on whether the external anal sphincter is perforated by the fistula tract in its cranial or caudal part, it is referred to as a "high" or "low" transsphincteric anal fistula. The high transsphincteric fistula is not clearly defined. Usually, a high transsphincteric anal fistula is referred to when >30% of the external anal sphincter is affected, i.e., the fistula traverses the muscle in its upper two-thirds.